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Lead poisoning and lead absorption

Chapter 26: REFERENCES.
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The authors survey lead’s chemistry, historical uses, and the routes and mechanisms of absorption, emphasizing inhalation of dust in industrial settings. They examine susceptibility, statistical occurrence, pathological changes, nervous system effects, methods for detecting and measuring lead, excretion, clinical symptoms, and treatment. Detailed experimental findings are presented to clarify causation and inform prevention, and practical recommendations focus on engineering controls such as local exhaust ventilation and workplace hygiene. Descriptions of hazardous processes and discussion of regulatory and technical measures aim to translate clinical and laboratory knowledge into strategies for reducing occupational lead risk.

Occupation. Causes of Death.
All
Causes.
Alco-
holism.
Gout. Phthi-
sis.
Diseases
of the
Nervous
System.
Diseases
of the
Circu-
latory
System.
Diseases
of the
Respi-
ratory
System.
Diseases
of the
Diges-
tive
System.
Bright’s
Disease.
Other
Diseases
of the
Urinary
System.
Plumbism. Acci-
dent.
(1) (2) (3) (4) (5) (6) (7) (8) (9) (10) (11) (12) (13)
All males 1,000 16 2 186 105 145 174 57  35 17   1 59
Printer   994  8 3 300 111 125 131 55  42 15   2 21
File-maker 1,700 14 387 225 198 325 78 134 26  56 46
Copper-worker 1,090  7 3 162 104 139 357 45  24 21   3 51
Lead-worker 1,408 38 165 134 222 309 14 160 102 52
Coach-maker   824  4 4 129 113 129 150 46  39 14   8 29
Earthenware 1,493  8 285 131 219 473 57  33 20  10 33
Glass 1,260  7 4 283 131 177 268 54  58 16   8 31
Painter and plumber 1,114 13 8 213 133 105 168 31  74 20  23 50

Other conditions which might readily be admitted as sequelæ are optic neuritis, following on an attack of encephalopathy. No general statement can be made in regard to mental and nervous diseases, gout, pernicious anæmia, as sequelæ, as each must be considered in relation to the evidence adduced in the particular case, and after exclusion, in the first two, of syphilis as a cause.

The distinction between causation and association has to be borne in mind before admitting as sequelæ of lead poisoning diseases of bacterial origin, such as phthisis or pneumonia, or any disease to which the affected person may be thought to have been rendered more prone by reason of lead employment. The contention that a person may have been debilitated by lead poisoning is no proof that the enfeeblement of the constitution was the cause either of the bacillus gaining entrance into the lung or of the ultimate fatal issue from the engrafted disease. Such assertion in every case must rest on supposition. Evidence that lead employment predisposes to phthisis is not necessarily made stronger, in our opinion, by existence during life of clinical symptoms, or, in their absence, of detection of lead in the tissues post mortem.

In classifying causes of death, the general rule should be to select, from the several diseases mentioned in the certificate, the disease of the longest duration. Exceptions to this rule are that definite diseases ordinarily known as constitutional diseases should have preference over the other diseases mentioned. After thirty-five years of age, certificates of death from lead poisoning are almost always filled in in association with other diseases which are the usual causes which lead to mortality generally. But neither phthisis, nor pneumonia, nor any acute disease of the heart or lungs, nor valvular disease of the heart, nor, indeed, any acute febrile condition, can have direct relation with—i.e., be a sequela of—lead poisoning.

REFERENCES.

[1] Annual Reports of the Chief Inspector of Factories since 1898, especially for 1909, p. 19.

[2] Supplement to the Sixty-fifth Annual Report of the Registrar-General on the Mortality in Certain Occupations in the Three Years 1900, 1901, 1902, by Dr. John Tatham, pp. cxix-cxxii, Cd. 2619.