WeRead Powered by ReaderPub
Shell-shock and other neuropsychiatric problems cover

Shell-shock and other neuropsychiatric problems

Chapter 262: Case 237. (Elliot, December, 1914.)
Open in WeRead

About This Book

The work assembles nearly six hundred clinical case histories drawn from wartime medical literature to document combat-related neuropsychiatric disorders. It presents concise case protocols illustrating varied symptom patterns, diagnostic dilemmas, malingering and simulation, therapeutic interventions, and treatment outcomes, and includes bibliographic references and introductory commentary. Sections juxtapose cases to illuminate contested diagnoses and to inform postwar rehabilitation and mental-hygiene efforts, aiming to provide clinicians and reconstruction workers with detailed clinical material for recognizing, classifying, and managing neuropsychiatric consequences of war.

Shell-shock; burial; flexion of spine: Paraplegia.

Case 237. (Elliot, December, 1914.)

A reservist, 34, formerly army instructor in gymnastics, a member of the 1st Battalion King’s Royal Rifles, was subject to injury from the bursting of a “Black Maria” on his trench. He was sitting with bent back in his shelter, with legs fully extended. He was in a small dug-out, a recess excavated under the earth backward from a narrow trench and not timbered. The “Black Maria” burst and covered him up to the chin in a heavy clay soil. After building up the breach twenty minutes later, his comrades dug him out.

He had received on his body the violent impact of the mass of earth pushed laterally from the crater excavated by the bursting of the shell. Accordingly his vertebral column was forcibly flexed, its ligaments were stretched, and hemorrhages were produced in the great muscles of the back. As the twelfth thoracic vertebra is the weakest spot in the spine, the roots of the cauda equina opposite this weak spot were probably injured. Such accidents are met in mines.

The legs were powerless and numb. There was nausea, no vomiting, no gas, no dizziness or trouble in the head, not even pain in the small of the back. The accident had occurred at 8 A.M. Upon nightfall, he was removed on a stretcher to the field hospital, arriving at the base hospital four days later; and on the fifth day power began to return to the legs. Knees, ankles, and toes would move slightly November 6, though passive movements of the legs caused pain in the back. The deep reflexes were weak, the plantar reflexes flexor. The left cremasteric reflex was weaker than the right. Impairment of sensation was slight in both extremities, but the left leg was a little more numb than the right. The left lower abdominal reflex was lost. A band of hyperalgesia corresponded with the left eleventh and twelfth thoracic segments November 12, slight reflex disorders and some degree of paresis of the legs.