Etiology and Occurrence.—Rupture of the flexor tendons or of the suspensory ligament is of rare occurrence. Frequently, these structures are divided as the result of wounds; but rupture, due to strain, is not frequent.

With rupture of the deep flexor tendon (perforans), especially when it occurs at or near its point of insertion and sometimes following disease, prognosis is unfavorable.

Rupture of the suspensory ligament constitutes a condition which is, as a rule, hopeless, because of the impracticability of treating such cases.

The salient feature which characterizes any practical attempt at treatment of ruptured tendons or other portions of the inhibitory apparatus of the fetlock region, is to retain the phalanges in their normal position for a sufficient length of time that the approximated ends of ruptured tendons or ligaments may unite. The length of time required for this to occur, together with the difficulties encountered in confining the affected extremities in suitable braces or supportive appliances, precludes all possibility of this condition's being practically amenable to treatment when the deep flexor tendon (perforans) and suspensory ligament are simultaneously ruptured. It does not follow, even so, that recovery does not succeed treatment in some of these unfavorable cases.

Affected subjects are kept in slings as long as it seems necessary—until they learn to get up without deranging the braces worn.

Several styles of braces are in use and each has its objections; nevertheless some sort of support to the affected member is necessary and steel braces which are connected with shoes are usually employed.

The principal difficulty which attends the use of braces is pressure-necrosis of the skin which is caused by the constant and firm contact of the metal support. The practitioner's ingenuity is taxed in every case to contrive practical means of padding the exposed parts in order to prevent or minimize necrosis from pressure. This is attempted—with more or less success—by frequent changing of bandages and the local application of such agents as alcohol or witch hazel. Needless to say, the skin must be kept perfectly clean and the dressings free from all irritating substances.

Etiology and Occurrence.—Synovial distension of tendon sheaths and bursae in the region of the fetlock are caused by the same active agencies which produce this condition in other parts. The fetlock region is exposed to more frequent injury than is the carpus and as a consequence is more often affected. The same proportionate amount of irritation affects this part of the leg, owing to strains, as affect the carpus from a similar cause; and synovitis from this cause, is as frequent in one case as in the other. Therefore, it is a natural sequence that the tendon sheaths of the metacarpophalangeal region are frequently distended because of chronic synovitis and thecitis. These inflammations are usually non-infective in character.

The cul-de-sac of the capsular ligament of the fetlock joint which extends upward between the bifurcation of the suspensory ligament is the most frequently affected structure in this region. When distended, two spheroidal masses bulge laterally and anterior to the flexor tendons in a characteristic manner. This condition is known among horsemen as "wind-gall" or "fetlock-gall."

The sheath of the flexor tendons, which begins about the middle portion of the lower third of the metacarpus, and continues downward below the pastern joint is often distended.

Excepting in cases of acute inflammation attending synovitis of these parts, no lameness marks its existence and in chronic cases of synovial distension the service of affected animals is not interfered with. These distensions constitute unsightly blemishes and they are treated chiefly for this reason.

No difficulty is encountered in recognizing these conditions even where considerable organization of tissue overlying distended thecae occurs. In such cases there may be only slight fluctuation of the enlargement, but if necessary, an aseptic exploratory puncture may be made with a suitable needle or trocar.

Treatment.—Complete rest and the local application of cold packs are in order in acute synovitis when there is distension of tendon sheaths. In the fetlock region, because of the ease with which pressure may be employed, the parts should be kept snugly wrapped with cotton, and derby bandages are used to exert the desired amount of pressure over the affected region. The pressure-bandages should be employed as soon as all acute and painful inflammation has subsided; and then they should be continued, day and night, for ten days or two weeks. The bandages should be removed morning and night. After the skin of the leg has thoroughly dried off, an infriction of alcohol or distilled extract of hamamelis is given the parts and the cotton and bandages are readjusted. A good, even and firm pressure in such cases is productive of satisfactory results.

Where no marked swelling occurs within ten days, as the result of the injection of iodin, the injection may be repeated and, if thought necessary, the quantity may be materially increased. If swelling does not occur it is indicative that no particular irritation has been caused.

Some swelling is desirable and much swelling sometimes results and persists for weeks. This is not in any way likely to cause permanent trouble; and if the technic of injection is skilfully executed no infection will follow.

By persistent and careful use of suitable elastic bandages, the support thus given the parts, together with the absorption of products of inflammation which constant pressure occasions, some chronic cases of synovial distension of tendon sheaths recover in two or three months and this without other treatment. Such good results are not to be expected in aged subjects, nor in horses having at the same time, chronic lymphangitis.

Where bandages of pure rubber are employed great care is necessary, if one is not experienced in their use, lest necrosis result. Where bandages are uncomfortably tight the subject will manifest discomfort, and an attendant should observe the animal at intervals for a few hours (where there may be some doubt as to the degree of pressure which is exerted by elastic bandages) and readjustment made before any harm is done.

In cases where there exists a synovitis or where a very limited portion of the articulation is involved, a somewhat different clinical picture is presented. Then, the disturbance causes less distress; local swelling and evidence of supersensitiveness are not so pronounced and lameness is not intense, though weight-bearing is painful.

Prognosis.—There is a constant difference in the degree of pain manifested, as well as the other symptoms of inflammation, between true arthritis, which involves much of the joint, and synovitis; or synovitis plus a small circumscribed area of joint involvement. This difference is present in all joint affections of the extremities and, in passing, it is well to say that infection usually increases every manifestation of pain. Infection occasions more pronounced local symptoms of inflammation and, because of the rapid progress of necrotic destruction of cartilage, the course of the affection is usually rapid; ankylosis is a frequent result and loss of the subject is often inevitable. However, in non-infective arthritis of the fetlock joint, prognosis is favorable.

Treatment.—The same general principles which are employed in arthritis of other joints are used here. Rest and comfort for the patient is sought in every available manner. If the subject remains standing too long, the sling should be used and a well-bedded box-stall will contribute much to the comfort of the patient.

Pain and acute inflammation is diminished or controlled, if possible, by using ice-cold packs. In nervous, well-bred animals analgesic agents may be employed; or small doses of morphin sulphate—one to two grains—given at intervals of three hours during the first stages of the affection is very beneficial. This is especially indicated in infectious arthritis.

After-care.—In the course of three or four weeks subjects may be allowed the run of a paddock and, after a complete rest of six weeks at pasture, they may be returned to work with care, if the work is not of a nature to occasion concussion or other manner of irritation to the articulation.

Neurectomy is not indicated even though there is a recurrence of lameness, unless the lameness is not pronounced and inflammation is periarticular and no osseous enlargements mechanically interfere with function of the joint. There are few cases then, where neurectomy is materially helpful.

Anatomy and Function of the Cartilages.—Surmounting each wing of the distal phalanx (os pedis) is the irregularly-quadrangular cartilage. The superior border of this cartilage is thin, generally convex, and perforated for vessels to pass to the frog; the inferior border is attached to the wing of the third phalanx and posteriorly, it is reflected inward and is continuous with the inferior surface of the sensitive frog. The anterior border which is directed obliquely downward and backward becomes blended with the anterior lateral ligament of the coffin joint. The fibrous expansion of the anterior digital extensor (extensor pedis) is united to the anterior borders of the lateral cartilages.

Symptomatology.—Ossification of the cartilages is known by grasping the free borders with the fingers and attempting their flexion; the rigid inflexible ossified cartilage is thus easily recognized.

Lameness during weight-bearing occurs in the majority of cases at some time. Much depends on the conformation of the foot and whether the involvement affects one or both cartilages as to the degree and duration of lameness which marks this affection. In narrow and contracted heels it is reasonable to expect more lameness than in well formed feet. Where only one cartilage is ossified, the other being flexible, there is less inconvenience experienced by the subject during weight-bearing, because of the expansion of the heel which the one normal cartilage allows.

Treatment.—There is little if anything to be done in case the cartilage has become ossified except to shoe without high calks but preferably with rubber pads. The hoof should be kept moist; the wall at the quarter may be rasped thin and kept anointed. Firing is of no practical benefit in these cases, and it is doubtful if vesication is helpful excepting where only a part of the cartilage is ossified.

Subjects which continue somewhat lame, because of complete ossification of both cartilages, are best put to slow work on soft ground and not driven on pavements.

This more or less ambiguous term has been applied to various diseases affecting the structures which make up the coffin joint. We consider this name to be applicable to inflammatory involvement of the third sesamoid (navicular bone), the deep flexor tendon (perforans) and the bursa podotrochlearis or navicular bursa.

Often both fore feet are affected and this would point toward its being a disease wherein either conformation or congenital tendencies exists. It is rare that hind feet are involved.

There are many theories regarding the possible exciting causes of navicular disease and, when one has carefully considered the explanations as offered by Peters, Möller, Branell, Schrader and others, he may conclude that navicular disease is a non-infectuous inflammatory affection of the third sesamoid (navicular) bone, deep flexor tendon (perforans) and adjoining structures. Whether it originates in the flexor tendon or whether the bone is the original part affected, the disease is frequently met, and of all possible causes, jars and irritation incident to concussion of travel, are probably the principal causative agents.

Symptomatology.—Lameness is the primary indicator and a constant symptom which attends navicular disease wherever much structural change affects the infirm parts. As the degree of intensity or extent varies, so is there a dissimilarity in the character of the impediment. Incipient cases of bilateral involvement are more difficult to diagnose than are unilateral affections, particularly when lameness is not marked. There is manifested a supporting-leg-lameness which varies as to degree in the same subject at different times. This may be noticed during the same trip in an animal that is being driven. There is a tendency for the subject to stumble and, of course, where the affection is bilateral, there is a stilted gait owing to shortened strides.

At rest the lame animal usually points with the affected member. Because of the fact that the distance is lessened between the origin and insertion of the deep flexor tendon (perforans) by this attitude, one may readily understand the reason for the position assumed by the subject. Pressure on the navicular bone is diminished and tension on the flexor tendon is relieved by even slight volar flexion.

In acute inflammatory affections abnormal heat may be detected in the region of the heel. By exerting tension on the flexor tendon, by means of passive dorsal flexion of the member, evidence of hyperesthesia may be detected. With the hoof testers one may determine supersensitivenss in most instances. There occurs more or less contraction of the hoof in navicular disease, but this is not to be taken as a cause of the affection, but rather a sequence.

Treatment.—When the anatomy of the diseased parts is taken into consideration, and an analysis of the lesions which occur in cases where considerable structural change is occasioned by this affection, it is obvious that recovery is impossible. Only in cases where the inflammation is promptly checked before damage has been done the navicular bone or the flexor tendon, is permanent recovery possible. The disease is not frequently treated during this stage, however, and in the majority of instances the condition becomes chronic.

As soon as a diagnosis is made the shoes must be removed, the toe shortened with the hoof pincers and rasp and the subject is put in a well bedded box-stall. If the animal is very lame and the inflammation is acute, ice-cold packs should be applied to the feet. As soon as acute inflammation has subsided the foot may be so pared that all excess of sole and frog is removed without lowering the heels, and the animal may be blistered about the coronet region. The subject may be shod later, with heel calks that raise the heel moderately and a protracted period of rest should be enforced.

In cases where no acute inflammatory condition exists, neurectomy is beneficial. One must discriminate, however, between favorable and unfavorable subjects. This is not a last resort expedient to be employed in cases where extensive lesions of the navicular structures exists. With proper shoeing, and by putting the subject at suitable work, where concussion of fast travel on hard roads is not necessary, the best results are obtainable.

This disease is primarily a non-infective inflammation of the sensitive laminae which very frequently affects the front feet. Often all four feet are affected, less frequently one foot (when its fellow is unable to sustain weight) and rarely the hind feet alone.

This disease occurs in connection with digestive disorders of various kinds and, because of the frequent association of the two conditions, the common term "founder" has long been employed to designate laminitis. In cases of "over-loading," particularly when a large quantity of wheat has been eaten by animals that are unaccustomed to this diet, laminitis almost constantly results.

Large draughts of cold water, when drunk by animals that are overheated is often followed by laminitis. Concussion, such as attends hard driving, especially in unshod horses or on rough and hard roads, is often succeeded by this affection. Likewise, as has been stated, injury such as is occasioned by long continued standing on the same foot is followed by laminitis. Some horses that are frequently shod, suffer from this affection a few hours after shoes have been reset. Dr. Chas. R. Treadway of Kansas City reports the rather frequent occurrence of such conditions in horses that are in the fire department service in his city.

Age in no way influences the occurrence of laminitis and the general condition of an animal with regard to its vigor or state of flesh has no apparent influence toward predisposing horses to this ailment.

This grouping does not include the effect of direct injuries of any and all kinds to which the feet are subjected such as: Concussion in fast road work, injuries occasioned by tight or ill fitting shoes, contusions of any kind resulting in non-infectious inflammation of the sensitive laminae, as well as the causes which produce laminitis where weight is borne by one foot when its fellow is out of function.

A classification which is practical is that of acute and chronic laminitis. To the practicing veterinarian it is this manner of consideration that is essential in the handling of these cases.

When the subject is caused to walk, symptoms of excruciating pain are manifested in all acute cases of laminitis. In some cases where all four feet are affected, no reasonable amount of persuasion will cause the suffering animal to move from its tracks.

There is acceleration of the rate of heart action; the pulse is full and in some cases, bounding. As the affection progresses the pulse becomes rather weak and irregular. The character of the pulse in the region of the extremity is a reliable indicator; but one has to learn to make necessary discrimination because of the condition of the parts, as in some cases of lymphangitis or where the skin is abnormally thick. The characteristic throbbing pulse is, however, easily recognized in most cases. Temperature is variable, though usually elevated from one to four degrees above normal. This symptom varies with the type and stage of the affection. In a subject that has been down, unable to rise for several days, where there is a suppurative and sloughing condition of the laminae, the temperature is high. Whereas, in some other and less destructive cases there may be little thermic disturbance after the first few hours have lapsed.

When we consider that the sensitive parts of the foot are encased by a horny, unyielding box and that, when the laminae are congested, a great pressure is brought to bear upon the sensitive structures, it is easy to understand why the condition is so painful.

Chronic laminitis is a sequel of acute inflammation of the sensitive laminae. It varies as to intensity and the exact manner of its manifestation depends upon preëxisting disturbances.

In some mild cases of laminitis there are recurrent attacks wherein no particular structural change exists, and diagnosis is established chiefly by noting the character of the pulse at the bifurcation of the large metacarpal (or metatarsal) artery just above the fetlock. The same manifestation of pain is present when weight is supported by one foot, though in a lesser degree. There is less local heat to be detected by palpation than in the acute cases.

Chronic laminitis as it occurs following acute attacks which have resulted in structural changes of the foot, present the same symptoms just described and, in addition, the peculiar alterations in structure exist. When, owing to acute inflammation of the sensitive laminae, there has resulted necrosis of this sensitive tissue together with infiltration between the anterior surface of the distal phalanx (os pedis) and the contacting hoof, the lower portion of the distal phalanx is turned downward and backward (rotated upon its transverse axis). Because of the traction which is exerted by the deep flexor tendon (perforans), as it attaches to the solar surface of the distal phalanx, this rotation is facilitated. With hyperplasia of lamina, at the anterior portion of the distal phalanx, there results a thick "white line." Rotation of the distal phalanx necessitates a descent of its apical portion and there occurs a "dropped sole."

In time, partly because of excessive wear of hoof at the heel, owing to an altered condition in the normal antagonistic relation between the flexor and extensor tendons, the toe makes an excessive growth, and the concavity of the anterior line is accentuated owing to this abnormal length of hoof. The hoof, because of recurrent inflammatory attacks, is corrugated—elevations of horn in parallel rings are usually present.

The same authority may be profitably quoted in the matter of handling all cases wherein the revulsive effect of agents which diminish vascular tension are chiefly indicated or necessary as adjuvants. In this connection, Campbell says: