Fortunately, because of the heavy musculature which goes to form a part of the locomotive apparatus of the rear extremity, hip lameness is comparatively rare. While the term is in itself ambiguous and signifies nothing more definite than does "shoulder lameness," yet diagnosis of almost any condition that may be classed under the head of "hip lameness" is not easy except in cases where the cause is obvious, as in wounds of the musculature and certain fractures. To the complexity which the gait of the quadruped contributes, because of its being four-legged, there is added the complicated manner of articulation of the bones of the hind leg. This involves the hip in the manner of diagnostic problems and because of the inaccessibility of certain parts, owing to the bulk of the musculature of these parts, diagnosis of some hip ailments becomes an intricate problem. Consequently, in some instances, before one may arrive at definite and enlightening conclusions, repeated examinations are necessary as well as a knowledge of reliable history and recorded observations of the subject over a considerable period.

Rheumatic affections, when present, usually cause recurrent attacks of lameness; myalgia, due to subsurface injury occasioned by contusion, generally produces an ephemeral disturbance; and while these are examples of cases where occult causes are active, they are by no means unprecedented. In cases where the cause of lameness is not definitely located, and when by the process of exclusion one is enabled to decide that the seat of trouble is in the hip, a tentative diagnosis of hip lameness is always appropriate.

Injuries of all kinds are the more frequent causes of hip lameness. In such cases, lameness may result directly and resolution be prompt, or the claudication become aggravated in time, due to muscular atrophy or degenerative changes affecting the hip joint or nerves. Rheumatism or metastatic infection may be the cause of hip lameness as well as affections of the pelvic bones, lumbar and sacral vertebrae. Hip lameness may also be provoked by melanotic or other tumors.

In the diagnosis of hip lameness, one is guided in a general way by the character of the impediment manifested. Swinging-leg lameness is often present and the impediment is more accentuated when the animal is caused to step backward. In many cases lameness is mixed, being about equally noticeable during weight bearing and while the member is being swung. By exclusion of causes which might affect other parts; one may definitely locate the cause of the trouble or determine that a certain region is affected.

The sudden manifestation of lameness is indicative of injury; thermic disturbances may signalize metastatic infection; history, if dependable, is always helpful. Repeated observations, taking into account the course which the affection assumes during a period of a few days, often serve to afford a means of establishing a diagnosis in baffling cases.

Etiology and Occurrence.—Pelvic fractures are usually caused by falls or other manner of contusion. Cases are reported where it would seem that fracture of the iliac angle resulted from muscular contraction, but it is certain that most fractures of this kind are due to collisions with door jambs or similar injuries. In old horses especially, fracture of pelvic bones occurs frequently. This form of injury is of more frequent occurrence in animals of all ages that work on paved streets. The country horse is not subjected to the uncertain footing of the slippery pavement, nor to injuries which compare with those caused by contusions sustained in falling upon asphalt or cobble-stones.

Symptomatology.—While in many cases of pelvic fracture lameness or abnormal decumbency are the salient manifestations, yet the pathognomic symptoms are crepitation or palpable evidence which may be obtained by rectal or vaginal examination. In fractures of the angle of the ilium and the ischial tuberosity, perceptible evidence always exists.

In cases where fracture of some portion of the pelvic girdle is suspected and the subject is able to walk, crepitation is sought by placing one hand on an external angle of the ilium and the other on the ischial tuberosity and the animal is then made to walk. Or, by placing the hands as just directed, an assistant may grasp the horse's tail and by alternately exerting traction on the tail and pushing against the hip in such manner that weight is shifted from one leg to the other, crepitation may be detected.

A regimen that is nutritive and at the same time laxative is essential and in some cases cathartics and enemata are necessary. Also, during the first few days, if there is retention of urine, catheterization is imperative. In a word, the handling of such cases consists largely in keeping the subject inactive, as comfortable as possible, and giving attention to suitable diet.

Simple fracture of the external iliac angle needs no particular attention, except that the subject is kept quiet until lameness subsides. In all cases where much of the bone is broken, the animal is blemished, but interference with function does not follow. If infection results because of a compound fracture, loose pieces of bone must be removed surgically and drainage provided for.

In fracture of the ischial tuberosity, infection is more apt to result than in like injury of the ilium, and greater displacement of bone occurs. This displacement, due to contraction of the attached muscles, is in some instances a contributing cause to the infection which often follows in these cases. In females where the body of the ischium is fractured, lacerations of the vagina may be present, and this constitutes a serious complication which usually terminates fatally.

After-care in fracture of the pelvic girdle consists principally in allowing a protracted period of rest before subjects are put to work.

Fracture of the trochanter major is signalized by local swelling and evidence of pain; the forward stride is shortened because this movement tenses the tendon of the gluteus major (maximus) which is attached principally to the trochanter.

Treatment.—When it is evident that a subject should be given treatment and not destroyed, the animal must be cast and completely anesthetized. With complete relaxation thus secured by rotation of the limb, using the hip joint region as a pivot, reduction may be effected. Traction is exerted in the same direction from the acetabulum that the head of the femur is situated and by pressing over the joint, the displaced bone may be returned in position. If luxation is downward, traction on the extremity will tend to dislodge the head of the femur from the inferior acetabular margin making reduction possible.

The same general plan which is ordinarily employed in correcting luxation is indicated here, but because of the heavy musculature of the hip, complete anesthesia is imperative in all such manipulations.

The glutens medius (g. maximus) muscle is inserted chiefly by means of two tendons; one to the summit of the trochanter major of the femur and the other passing over the anterior part of the convexity of the trochanter, and being attached to the crest below it. The trochanter is covered with cartilage, and a bursa (the trochanteric) is interposed between the tendon and the cartilage.

Etiology and Occurrence.—This affection is probably caused in most instances by direct injury to the parts, such as may be occasioned by being kicked, falling on pavement, or being struck by the body of a heavy wagon. Strains in pulling or in slipping are undoubtedly causative factors and in draft horses such strains may result in involvement of this synovial apparatus.

Treatment.—In the first stages of an acute affection absolute quiet must be enforced; local antiphlogistic applications are beneficial. Later, vesication of a liberal area surrounding the trochanter major is indicated. Where the condition has become chronic in horses that are to be kept at heavy draft work there is little chance for complete recovery. And, naturally, one is not to expect resolution in cases where there exist erosion and ossification of cartilage—where crepitation is discernible.

Aside from paraplegic conditions due to disease of the cord or the lumbosacral plexus, and monoplegic affections resultant from disturbances of this plexus, paralysis of certain nerves are occasionally encountered.

Etiology and Occurrence.—While paralysis of the femoral nerve, also known as "dropped stifle" occurs as a result of local injuries and melanotic tumors in gray horses, most cases are due to azoturia. So-called crural paralysis or "hip swinney" is occasionally observed but this is not a condition wherein the nerve is affected in the manner that characterizes the marked atrophy of quadriceps femoris (crural) muscles in some cases of hemaglobinuria. This form of paralysis according to Hutyra and Marek is due primarily to diffuse degeneration of the muscles.

Symptomatology.—When muscular atrophy is not extensive no particular evidence of this condition may be manifested while the subject is at rest, but where muscular waste has occurred, the nature of the ailment is at once recognized. Since the femoral nerve supplies the quadriceps femoris muscles, it follows that when the psoic portion of this nerve becomes diseased, the stifle loses its support, and in a unilateral involvement when the subject attempts to walk on the affected member, the stifle sinks down for want of support and the leg collapses unless weight is caught up with the other leg. Often, following azoturia, a bilateral affection is to be observed.

Treatment.—Horses may be restrained in the standing position, and in the average instance, a twitch and hood are all the restraining appliances necessary.

In cases where the disease is unilateral and atrophy is not of too long standing, recovery is possible in vigorous subjects. All affections, however, wherein degenerative changes involve the nerve trunk, whether due to diffuse myositis or pressure from malignant tumors, will not yield to treatment.

In addition to the foregoing, good results have attended the use of intramuscular injections of oxygen. The technic of the operation consists in preparing the area of skin which covers the atrophied muscles as for any operation. The hair is clipped over five or six or more circular areas of about an inch in diameter; the skin is cleansed and then painted with tincture of iodin.

A long heavy sterile needle, which is connected with an oxygen tank by means of six feet of rubber tubing, is thrust into the depths of the affected muscles and the gas is gently introduced into the tissues. One needs exercise extreme care that the gas enter slowly because great pain is produced by the sudden injection of the oxygen. Likewise too much of the gas must not be introduced at one place. When the oxygen is slowly introduced it may be allowed to enter the tissues until the subject gives evidence of experiencing considerable pain, or if the parts are not particularly sensitive, a reasonable amount (enough to cause a mild degree of diffuse inflammation) is introduced at each one of five or six points. In large animals more points of injection may be used.

No infection or other bad results will follow the execution of a good technic and the treatment may be repeated every three or four weeks until either marked regeneration of tissue is evident or the case is obviously proved hopeless.

Anatomy.—The obturator nerve, situated at first under the peritoneum, accompanies the obturator artery through the obturator foramen and gaining the muscles on the internal face of the thigh, terminates in the obturator externus, adductors, pectineus and gracilis, also giving twigs to the obturator internus (Strangeways).

Symptomatology.—In a unilateral affection there may be little evidence of the trouble while the subject is standing; or there is to be seen some abduction; or the affected member may present abduction of the stifle and stand "toe outward." If the animal is walked there will be manifested more or less abduction and the character of the impediment varies according to the nature of the involvement.

Following protracted cases of labor in some instances where only a unilateral paralysis exists, walking is performed with difficulty; the subject may be unable to support weight with the affected member and is obliged to hop on the one sound hind leg. In bilateral affections, they are unable to rise. If the condition is severe the sling is required to keep the subject standing, and with this care, recovery will follow.

Treatment.—If new growths or callosities or similar conditions affect the nerve, little, if any, hope for recovery exists. In young and vigorous subjects where cause is not definitely known, a course of strychnin may be given. Good nursing, providing for the subject's comfort and allowing moderate exercise, constitute rational treatment. Stimulating embrocations on the abductor muscles resorted to in cases during the incipient stage may prove helpful.

Good hygienic care, a suitable diet and full physiological doses of strychnin are indicated. Cadiot and Almy recommend vaginal douches of cold water and counterirritation of the region of the inner thigh in these cases.

Anatomy.—The great sciatic nerve leaves the pelvis in company with the gluteal nerves, through the great sciatic foramen (notch), passing downward along the posterior face of the femur. Near the stifle it passes between the two heads of the gastrocnemius muscle and continues as the tibial. Branches supply the following muscles—obturator, semimembranosus (adductor magnus), biceps femoris (triceps abductor femoris), semitendinosus (biceps rotator tibialis), lateral extensor (peroneus) and the tibial nerve, its continuation, innervates the digital flexors.

Etiology and Occurrence.—Paralysis of the great sciatic nerve may be caused by central disorders, injury in falling, fractures and new growths. Because of its protected position, this nerve does not often suffer injury, and paralysis of the sciatic nerve is recorded in a few instances owing to its rarity.

Symptomatology.—When consideration is given the number of muscles that are supplied by the sciatic nerve and the function of these muscular structures, it is obvious that the leg cannot be used in sciatic paralysis. However, the limb is capable of sustaining weight when it is fixed in position, but this is done without exertion of muscular fibers which are supplied by the great sciatic nerve. Trotting is impossible and flexion of the affected member is also likewise precluded. The foot is dragged when the subject is caused to advance.

This was not a paralytic condition and recovery resulted, yet undoubtedly this was a case which, if not properly cared for, might have terminated unfavorably.

Treatment.—Prognosis is decidedly unfavorable in paralysis of the great sciatic nerve. If treatment is attempted, it is to be conducted along the same general lines as in femoral paralysis. Particular attention should be given to conditions which will make for the patient's comfort, and as soon as it is evident that the affection is not progressing favorably, the subject should be humanely destroyed.

This condition is undoubtedly of more frequent occurrence than we are wont to grant when one considers the comparatively small number of cases that are actually recognized in practice. It does not follow, however, that iliac thrombosis rarely exists. Probably in the majority of instances there is insufficient obstruction of the lumina of vessels to provoke noticeable inconvenience. Or, if circulation is hampered to the extent that function is impaired and manifestations are observed by the driver, the subject may be permitted to rest a few days and partial resolution occurs, so that further trouble is not noticeable.

As judged by lesions of the aorta and iliac arteries in dissecting subjects, the conclusion that arteritis and resultant disorders are of rather frequent occurrence, is logical.

Etiology.—Inflammation of the vessel walls and resultant prolifieration of tissue together with the accumulation of clotted blood becoming organized, serve to obstruct the lumen of the affected artery. The cause of arteritis is unknown in many instances, but parasitic invasion and contiguous involvement of vessels in some inflammatory injuries are etiological factors.

Etiology and Occurrence.—Patellar fractures are rarely met with in the horse but may be caused by falls and heavy contusions. Violent muscular contraction, it is said, may also bring about the same condition.

Symptomatology.—Fracture may be transverse or vertical, and depending on the manner in which the bone is broken, prognosis is either at once rendered favorable or unfavorable. The patella performs a function which is in a way similar to that of the sesamoids and when fractured, complete recovery is improbable in the average instance. When complete, transverse fractures permit of separation of the parts of bone. Tension on the straight ligaments below and contraction of the quadriceps above usually cause insuperable difficulty in the handling of this type of fracture in the horse.

Compound fractures as well as multiple or comminuted fractures occasionally occur and these constitute injuries which are generally considered fatal, although Andrien, according to Cadiot and Almy, succeeded in obtaining complete recovery in a case of compound fracture of the patella and the horse was in service and almost free from lameness two months after treatment was begun.

No difficulty is encountered in recognizing the fracture of the patella because of the exposed position of the bone. Crepitation, and in some cases fissures, may be easily detected.

Treatment.—In simple fracture, when treatment is thought advisable, the subject is put in a sling and kept as nearly comfortable as possible. If little inflammation exists, the application of a vesicant two or three weeks after the injury has been inflicted will be helpful and serve to hasten repair.

Bandages or mechanical appliances are of no practical use in the handling of these cases.

Etiology and Occurrence.—This, the most common luxation met with in the equine subject, has been described by writers as existing in many forms. Patellar disarticulation may be more practically considered as momentary and fixed, regardless of the position taken by the patella. Described under the title of false luxation are recorded cases wherein the quadriceps (crural) muscles become contracted in such manner that a condition simulating true disarticulation of the patella obtains. Also, some practictioners report cases of patellar luxation and refer to pseudo-luxations, without clearly defining the conditions which constitute pseudo-luxation. This has contributed to the extant cause of misconception as to actual differences between luxation and conditions simulating dislocation.

Luxation of the patella is a condition wherein the articular portions of the femur and patella assume abnormal relations whether such displacement of the patella be momentary and capable of spontaneous reduction, or fixed and requiring corrective manipulation. Spasmodic contraction of the crural muscles which sometimes retains the patella in such position that the leg is rigidly extended, does not in itself constitute luxation of the patella; and unless this bone becomes lodged on the upper portion of a femoral condyle or laterally displaced out of its femoral groove, luxation cannot be said to exist in the horse. These are sub-luxations.

Occasionally one may observe in suckling colts outward luxation of the patella wherein there is history of navel infection and no marked evidence of rachitis is present. Some of these cases recover. In a unilateral involvement of this kind in a three-month-old mule colt, the author observed a case wherein an unfavorable prognosis was given and destruction of the subject advised, because of the extreme dislocation of the patella. This colt, however, was not destroyed and in three weeks had apparently recovered. No treatment was given in this instance; the colt was allowed the run of a small pasture with its dam and in time it matured, becoming a sound and serviceable animal.

The first form is known as upward luxation and is made possible by rupture of the mesial (internal) femeropatellar ligament. According to Cadiot and Almy, it is only by the rupture of this ligament—the femeropatellar—that upward luxation may occur. This type of luxation is rarely observed and is usually due to violent strain and abnormal extension of the stifle joint.

The second class, outward luxation, occurs in colts and is, in many instances, congenital. This form of luxation is also the one usually seen following debilitating diseases such as influenza and pneumonia.

Upward luxation of the patella is characterized by the stiff-extended position of the leg. When the patella is situated upon the inner trochlear rim, the tibia must be extended because of the traction exerted by the straight ligaments. Since the stifle and hock joints extend and flex in unison, there is presented also an extension of the tarsus. Extension of the stifle joint would increase the distance between the femoral origin of the gastrocnemius and its insertion to the summit of fibular tarsal bone (calcis) were it not for the gastrocnemius and superficial flexor (perforatus). Extension of the hock in upward luxation of the patella, permits of flexion of the phalanges. In upward luxation, then, the leg is extended as if too long, but the phalanges may be in a state of moderate flexion. If the foot rests on the ground when the extremity is not flexed, it is almost impossible for the subject to step backward. Because of immobilization of the stifle and hock joints in upward luxation, the subject can walk only by hopping on the sound leg and then the extremity is flexed, allowing the anterior portion of the fetlock to drag on the ground.

In upward luxation, reduction is effected by attempting further extension of the stifle joint and at the same time the patella is pulled outward, off the internal rim of the trochlea. This is attempted by securing the subject in a standing position; the sound side is kept against a wall if possible and a rope is tied to the extremity of the affected leg. Traction is exerted upon the rope and at the same time force is directed against the stifle joint to produce further extension if possible, so that the straight patellar ligaments may relax sufficiently to allow the patella to be dislodged from its position upon the inner trochlear lip. Failing in this manner of procedure, the affected animal is to be cast and anesthetized with chloroform. The relaxation which attends surgical anesthesia will permit of reduction of the dislocated bone and manipulations such as have just been outlined may be employed.

Following reduction in the average case it is essential that the subject be given vigorous exercise for a few minutes. Reduction having been affected, the application of a vesicant over the whole patellar region is customary.

In cases of habitual luxation, unless the ligaments are so lax that the patella may be displaced laterally over the inner as well as the outer trochler rims, division of the inner straight patellar ligament will correct the condition. This desmotomy has been advocated by Bassi, and good results in appropriate cases have been reported by Cadiot, Merillat and Schumacher. This operation has been found a corrective in cases of outward luxation as well as those of upward dislocation of the patella when resorted to before the trochleae are worn from frequent luxation.

As a rule, the reduction of this form of luxation is not difficult. The patella may be pushed inward and into position without manipulation of the leg. Retention of the patella in position is a difficult problem. Bandaging is considered impractical and is not ordinarily done in this country. Benard, according to Cadiot and Almy, recommends bandaging with a heavy piece of cloth in which an opening is made through which the patella is allowed to protrude, and by turning such a bandage snugly about the stifle several times, the patella is held in position. This bandage should be kept in place for about ten days.

In young and rachitic animals outdoor exercise and a good nutritive ration for the subject are indicated. Hypophosphites in assimilable form may be beneficial, and vesication of the patellar region contributes to recovery.

Luxations attending some cases of influenza recover promptly when subjects are kept comfortably confined in roomy box-stalls. The administration of stimulative medicaments such as nux vomica and the application of an active blistering agent to the patella serve to hasten recovery. Dislocations in such cases are often bilateral and they are usually momentary. Reduction occurs spontaneously, as a rule, and the subjects are not occasioned much distress if they are kept quiet for a few days.

Upon manipulation of the patellar region, one is impressed with the fact that hyperesthesia does not exist in proportion to the pain manifested during locomotion. In some cases a gelatinous swelling is present and may be detected by palpating between the straight ligaments of the patella. Williams, Hughes, Merillat, Hadley and others have directed attention to the existence of floating masses (corpora oryzoidea) in the synovial capsule of this joint in gonitis, and as with all cases of arthritis, irreparable damage is often done the articular cartilages during the course of the ailment.

Treatment.—No effective method is as yet known which will control this condition during its incipiency. The disease progresses, and more or less damage is done the affected parts in the course of months or even years in some cases before subjects are rendered hopelessly crippled. When recognized early (before chronic gonitis exists) aspiration of the synovia and the injection of diluted tincture of iodin might prove beneficial in cases of synovial distension. Chronic gonitis is considered an incurable affection and as soon as subjects manifest evidence of distress from this condition they should by all means be taken from work. Firing and vesication have not been productive of beneficial results.

Anatomy of the Joint Capsule.—This joint capsule is thin and very capacious. On the patella it is attached around the margin of the articular surface, but on the femur the line of attachment is at a varying distance from the articular surface. On the medial side it is an inch or more from the articular cartilage; on the lateral side and above, about half an inch. It pouches upward under the quadriceps femoris for a distance of two or three inches, a pad of fat separating the capsule from the muscle. Below the patella it is separated from the patellar ligaments by a thick pad of fat, but inferiorly it is in contact with the femerotibial capsules. The joint cavity is the most extensive in the body. It usually communicates with the medial sac of the femerotibial joint cavity by a slit-like opening situated at the lowest part of the medial ridge of the trochlea. A similar, usually smaller, communication with the lateral sac of the femerotibial capsule is often found at the lowest part of the lateral ridge. (Sisson's Anatomy.)

Thus it is seen that because of its frequent communication with the other parts of this large synovial membrane, a wound which opens the external portion of the femerotibial capsule may be the cause of contamination and resultant infectious arthritis of the whole stifle joint. Because of the distance between the most dependent part of the femerotibial articulation and the summit of the patella, one may misjudge the exact location of the lowermost part of this portion of the capsular ligament of the stifle joint and thereby fail at once to appreciate the seriousness of calk wounds in this region.

Symptomatology.—The pathognomonic symptom of open stifle joint is the profuse escape of synovia, indicating perforation of the synovial capsule; by means of a probe the wound may be explored in a way that will clearly reveal the nature of the injury.